How is it that many schizophrenics identify certain instances of verbal imagery as hallucinatory? Most investigators have assumed that alterations in sensory features of imagery explain this. This approach, however, has not yielded a definitive picture of the nature of verbal hallucinations. An alternative perspective suggests itself if one allows the possibility that the nonself quality of hallucinations is inferred on the basis of the experience of unintendedness that accompanies imagery production. Information-processing models of “intentional” cognitive processes call for abstract (...) planning representations that are linked to goals and beliefs. Unintended actions - and imagery - can reflect planning disruptions whereby cognitive products do not cohere with concurrent goals. A model of schizophrenic speech disorganization is presented that postulates a disturbance of discourse planning. Insofar as verbal imagery can be viewed as inwardly directed speech, a consequence of such planning disturbances could be the production of unintended imagery. This link between the outward disorganization of schizophrenic speech and unintended verbal imagery is statistically supported by comparing the speech behavior of hallucinating and nonhallucinating schizophrenics. Studies of “borderline” hallucinations during normal, “goal-less” relaxation and drowsiness suggest that experiential unintendedness leads to a nonpathological variant of hallucinatory otherness that is correctable upon emerging from such passive cognitive states. This contrasts with the schizophrenic case, where nonconcordance with cognitive goals reinforces the unintendedness of verbal images and sustains the conviction of an external source. This model compares favorably with earlier models of verbal hallucinations and provides further evidence for a language production disorder in many schizophrenics.Short Abstract: How is it that many schizophrenics identify certain instances of verbal imagery as hallucinatory? This paper proposes that the critical feature identifying hallucinations is the experience of unintendedness. This experience is nonpathological during passive conscious states but pathological if occurring during goal-directed cognitive processing. A model of schizophrenic speech disorganization is presented that postulates a disturbance of discourse planning that specifies communicative intentions. These alterations could generate unintended verbal imagery as well. Statistical data are offered to support the model, and relevant empirical studies are reviewed. (shrink)

An underlying assumption of default-interventionist dual-process theory (DI-DPT) of reasoning is that humans tend to act as cognitive misers. Although miserly cognitive processing occurs when indiv...

Further tests of Amit's model are indicated. One strategy is to use the apparent coding sparseness of the model to make predictions about coding sparseness in Miyashita's network. A second approach is to use memory overload to induce false positive responses in modules and biological systems. In closing, the importance of temporal coding and timing requirements in developing biologically plausible attractor networks is mentioned.

It is the speed of the development and manufacturing cycle that appears as technical innovation or leadership.

Transcranial magnetic stimulation, EEG, and behavioral studies by our group implicate spurious activation of speech perception neurocircuitry in the genesis of auditory hallucinations in schizophrenia. The neurobiological basis of these abnormalities remains uncertain, however. We review our ongoing studies, which suggest that altered cortical coupling underlies speech processing in schizophrenia and is expressed via disrupted gamma resonances and impaired corollary discharge function of self-generated verbal thought.

Dictionary entry discussing the main moral and meta-ethical doctrines found in the works of James Griffin.

This is a theological adventure based on the thought of Charles Hartshorne. Its appearance at this time represents an attempt to begin anew in theology on the assumption that the abstract God of classical thinking is dead. Hartshorne's philosophy advances a God of concrete and changing reality, as opposed to the abstract, immutable and "dead" God image of the radical theologians. The author argues that the "Death of God" theology is no more than a recognition that Christian incarnation is impossible (...) given classical presuppositions; argues for disallowance of the traditional image and positing of a neo-classic, dipolar concept of God which surpasses the abstract and makes acceptable the belief in a Supreme Being. (shrink)

Multidisciplinary studies indicate that auditory hallucinations may arise from speech perception neurocircuitry without disrupted theory of mind capacities. Computer simulations of excessive pruning in speech perception neural networks provide a model for these hallucinations and demonstrate that connectivity reductions just below a “psychotogenic threshold” enhance information processing. These data suggest a process whereby vulnerability to schizophrenia is maintained in the human population despite reproductive disadvantages of this illness.

The present study examined the effects of thought suppression on sleep-onset mentation. It was hypothesized that the decrease of attentional control in the transition to sleep would lead to a rebound of a suppressed thought in hypnagogic mentation. Twenty-four young adults spent two consecutive nights in a sleep laboratory. Half of the participants were instructed to suppress a target thought, whereas the other half freely thought of anything at all. To assess target thought frequency, three different measures were used in (...) the wake state and mentation reports were repeatedly prompted by a computer at sleep onset. In support of the hypothesis, results revealed a reversal of target thought frequency at sleep onset: Participants instructed to suppress reported fewer target thoughts than did controls before falling asleep, but more target thoughts afterwards. (shrink)

We propose that the primary cause of schizophrenia is a pathological extension of synaptic pruning involving local connectivity that unfolds ordinarily during adolescence. Computer simulations suggest that this pathology provides reasonable accounts of a range of symptoms in schizophrenia, and is consistent with recent postmortem and genetic studies. NMDA-receptors play a regulatory role in maintaining and/or eliminating cortical synapses, and therefore may play a pathophysiological role.

In order to reach a better understanding of brain function, conceptual synergies linking empirical neurobiological studies and neurocomputational studies should be pursued. I describe an example of a potential synergy based on studies of neural network pruning. Simulations demonstrate that selective elimination of connections enhances the computational capacity of networks capable of temporal processing. These findings may shed light on the functional significance of postnatal neuro-developmental pruning of cortical connections that occurs in mammals.

Although Freud's merits may be readily acknowledged in the year of his 150th birthday, recent findings on repression-related phenomena cannot be accommodated by his classic conception, on which Erdelyi's theory is built. This point is illustrated by discussing the role of inhibitory processes. The unified theory of repression should be elaborated to generate falsifiable predictions on the reported phenomena.